Fig. 2From: The role of podocyte damage in the etiology of ischemia-reperfusion acute kidney injury and post-injury fibrosisGlomerular ultrastructure of kidneys on the 28th day after renal arterial injury. Control, Sham and AKI 20: No foot process effacement or flattening was seen. AKI 30: extensive foot process fusion was detected. AKI 40: extensive podocyte foot process stripping and GBM exposure were detectedBack to article page