Fig. 3From: The role of podocyte damage in the etiology of ischemia-reperfusion acute kidney injury and post-injury fibrosisExpression of Synaptopodin, Nephrin, CD2AP, and TRPC6 protein by immunohistochemical staining. AKI 20 group (mice underwent renal artery clamping for 20 min followed by contralateral nephrectomy 8 days after injury), AKI 30 group (mice underwent renal artery clamping for 30 min followed by contralateral nephrectomy 8 days after injury), AKI 40 groups (mice underwent renal artery clamping for 40 min followed by contralateral nephrectomy 8 days after injury). Scale bar =50 μmBack to article page