Fig. 3From: APOL1 renal risk variants exacerbate podocyte injury by increasing inflammatory stressAPOL1-B3 regulated proinflammatory signaling in vitro. Immortalized human podocytes were stimulated by IL-1β (15 ng/ ml, 3 h). The expression of the APOL1 splice variant was examined by qPCR, using V2–3 primers and V1 primers as indicated by arrows in the above exon schema. The graph shows the relative expression of IL-1β to GAPDH. (Student’s t test, p < 0.05)Back to article page